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biotinylated primary antibody targeting cd68  (Bio-Rad)


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    Structured Review

    Bio-Rad biotinylated primary antibody targeting cd68
    Figure 6 | Proteinuric kidney injury stimulates ileal macrophage production of vascular endothelial growth factor (VEGF)-C. (a) Puromycin aminoglycoside (PAN) increased ileal VEGF-C versus controls (Cont). (b) VEGF-C concentration in PAN lymph was lower, but total output of VEGF-C was significantly greater, in PAN versus Cont. (c) Double staining of ileum with VEGF-C (red) and cluster of differentiation (CD) 68 (green) showed a greater number of <t>CD68-positive</t> cells and <t>CD68-</t> and VEGF-C–positive cells (arrows) in PAN versus Cont. (d) Cultured macrophages exposed to isolevuglandin (IsoLG)–apolipoprotein AI (apoAI) expressed more VEGFC mRNA versus unmodified apoAI. In vivo, results are mean SD for 7 to 12 rats per group. In vitro, experiments were performed independently 3 times with 3 wells per treatment. To optimize viewing of this image, please see the online version of this article at www.kidney-international.org.
    Biotinylated Primary Antibody Targeting Cd68, supplied by Bio-Rad, used in various techniques. Bioz Stars score: 96/100, based on 1043 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/biotinylated primary antibody targeting cd68/product/Bio-Rad
    Average 96 stars, based on 1043 article reviews
    biotinylated primary antibody targeting cd68 - by Bioz Stars, 2026-03
    96/100 stars

    Images

    1) Product Images from "Kidney injury-mediated disruption of intestinal lymphatics involves dicarbonyl-modified lipoproteins."

    Article Title: Kidney injury-mediated disruption of intestinal lymphatics involves dicarbonyl-modified lipoproteins.

    Journal: Kidney international

    doi: 10.1016/j.kint.2021.05.028

    Figure 6 | Proteinuric kidney injury stimulates ileal macrophage production of vascular endothelial growth factor (VEGF)-C. (a) Puromycin aminoglycoside (PAN) increased ileal VEGF-C versus controls (Cont). (b) VEGF-C concentration in PAN lymph was lower, but total output of VEGF-C was significantly greater, in PAN versus Cont. (c) Double staining of ileum with VEGF-C (red) and cluster of differentiation (CD) 68 (green) showed a greater number of CD68-positive cells and CD68- and VEGF-C–positive cells (arrows) in PAN versus Cont. (d) Cultured macrophages exposed to isolevuglandin (IsoLG)–apolipoprotein AI (apoAI) expressed more VEGFC mRNA versus unmodified apoAI. In vivo, results are mean SD for 7 to 12 rats per group. In vitro, experiments were performed independently 3 times with 3 wells per treatment. To optimize viewing of this image, please see the online version of this article at www.kidney-international.org.
    Figure Legend Snippet: Figure 6 | Proteinuric kidney injury stimulates ileal macrophage production of vascular endothelial growth factor (VEGF)-C. (a) Puromycin aminoglycoside (PAN) increased ileal VEGF-C versus controls (Cont). (b) VEGF-C concentration in PAN lymph was lower, but total output of VEGF-C was significantly greater, in PAN versus Cont. (c) Double staining of ileum with VEGF-C (red) and cluster of differentiation (CD) 68 (green) showed a greater number of CD68-positive cells and CD68- and VEGF-C–positive cells (arrows) in PAN versus Cont. (d) Cultured macrophages exposed to isolevuglandin (IsoLG)–apolipoprotein AI (apoAI) expressed more VEGFC mRNA versus unmodified apoAI. In vivo, results are mean SD for 7 to 12 rats per group. In vitro, experiments were performed independently 3 times with 3 wells per treatment. To optimize viewing of this image, please see the online version of this article at www.kidney-international.org.

    Techniques Used: Concentration Assay, Double Staining, Cell Culture, In Vivo, In Vitro



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    biotinylated primary antibody targeting cd68  (Bio-Rad)
    96
    Bio-Rad biotinylated primary antibody targeting cd68
    Figure 6 | Proteinuric kidney injury stimulates ileal macrophage production of vascular endothelial growth factor (VEGF)-C. (a) Puromycin aminoglycoside (PAN) increased ileal VEGF-C versus controls (Cont). (b) VEGF-C concentration in PAN lymph was lower, but total output of VEGF-C was significantly greater, in PAN versus Cont. (c) Double staining of ileum with VEGF-C (red) and cluster of differentiation (CD) 68 (green) showed a greater number of <t>CD68-positive</t> cells and <t>CD68-</t> and VEGF-C–positive cells (arrows) in PAN versus Cont. (d) Cultured macrophages exposed to isolevuglandin (IsoLG)–apolipoprotein AI (apoAI) expressed more VEGFC mRNA versus unmodified apoAI. In vivo, results are mean SD for 7 to 12 rats per group. In vitro, experiments were performed independently 3 times with 3 wells per treatment. To optimize viewing of this image, please see the online version of this article at www.kidney-international.org.
    Biotinylated Primary Antibody Targeting Cd68, supplied by Bio-Rad, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/biotinylated primary antibody targeting cd68/product/Bio-Rad
    Average 96 stars, based on 1 article reviews
    biotinylated primary antibody targeting cd68 - by Bioz Stars, 2026-03
    96/100 stars
    		  Buy from Supplier

    Image Search Results


    Figure 6 | Proteinuric kidney injury stimulates ileal macrophage production of vascular endothelial growth factor (VEGF)-C. (a) Puromycin aminoglycoside (PAN) increased ileal VEGF-C versus controls (Cont). (b) VEGF-C concentration in PAN lymph was lower, but total output of VEGF-C was significantly greater, in PAN versus Cont. (c) Double staining of ileum with VEGF-C (red) and cluster of differentiation (CD) 68 (green) showed a greater number of CD68-positive cells and CD68- and VEGF-C–positive cells (arrows) in PAN versus Cont. (d) Cultured macrophages exposed to isolevuglandin (IsoLG)–apolipoprotein AI (apoAI) expressed more VEGFC mRNA versus unmodified apoAI. In vivo, results are mean SD for 7 to 12 rats per group. In vitro, experiments were performed independently 3 times with 3 wells per treatment. To optimize viewing of this image, please see the online version of this article at www.kidney-international.org.

    Journal: Kidney international

    Article Title: Kidney injury-mediated disruption of intestinal lymphatics involves dicarbonyl-modified lipoproteins.

    doi: 10.1016/j.kint.2021.05.028

    Figure Lengend Snippet: Figure 6 | Proteinuric kidney injury stimulates ileal macrophage production of vascular endothelial growth factor (VEGF)-C. (a) Puromycin aminoglycoside (PAN) increased ileal VEGF-C versus controls (Cont). (b) VEGF-C concentration in PAN lymph was lower, but total output of VEGF-C was significantly greater, in PAN versus Cont. (c) Double staining of ileum with VEGF-C (red) and cluster of differentiation (CD) 68 (green) showed a greater number of CD68-positive cells and CD68- and VEGF-C–positive cells (arrows) in PAN versus Cont. (d) Cultured macrophages exposed to isolevuglandin (IsoLG)–apolipoprotein AI (apoAI) expressed more VEGFC mRNA versus unmodified apoAI. In vivo, results are mean SD for 7 to 12 rats per group. In vitro, experiments were performed independently 3 times with 3 wells per treatment. To optimize viewing of this image, please see the online version of this article at www.kidney-international.org.

    Article Snippet: Double staining for CD68 and VEGF-C used citrate buffer for antigen retrieval, followed by biotinylated primary antibody targeting CD68 (1:10; BioRad).

    Techniques: Concentration Assay, Double Staining, Cell Culture, In Vivo, In Vitro